Chapter 1 creation (pages 1–10): J.R. Vane
Chapter 2 Ultrastructural association of the Alveolar?Capillary Unit (pages 11–36): Maya Simionescu
Chapter three Hypoxia and Pulmonary Vascular Endothelium (pages 37–60): Lynne Reid and Barbara Meyrick
Chapter four equipment for the research of Lung Metabolism (pages 61–83): H.F. Woods, A. Meredith, G.T. Tucker and J.R. Shortland
Chapter five Substrate usage by way of the Lung (pages 85–104): H. Datta, W.A. Stubbs and K.C.M.M. Alberti
Chapter 6 Inactivation of Monoamines via the Lung (pages 105–128): M.B.H. Youdim, Y.S. Bakhle and R R. Ben?Harari
Chapter 7 The destiny of Circulating Biologically lively Peptides within the Lungs (pages 129–145): Sergio H. Ferreira, Lewis J. Greene, Maria Cristina O. Salgado and E.M. Krieger
Chapter eight The Lung as a Generator of Prostacyclin (pages 147–164): R.J. Gryglewski
Chapter nine Interrelationships among Prostacyclin and Thromboxane A2 (pages 165–183): S. Moncada and J. R. Vane
Chapter 10 rules of Pulmonary Arachidonic acid Metabolism via Anti?Inflammatory Steroids (pages 185–201): R.J. Flower
Chapter eleven Slow?Reacting components and Their Structural Elucidation (pages 203–215): Priscilla J. Piper, Marwa N. Samhoun, J. R. Tippins, H. R. Morris and G. W. Taylor
Chapter 12 The Lung on the subject of Vasoactive Polypeptides (pages 217–237): Sami I. stated, Viktor Mutt and Ervin G. Erdos
Chapter thirteen Endocrine impacts on features of Lung Biochemistry (pages 239–250): Walter okay. Morishige
Chapter 14 Hormonal impacts in the course of Fetal Lung improvement (pages 251–274): Philip L. Ballard
Chapter 15 Pulmonary Angiotensin?Converting Enzyme and Its Inhibition: A ancient Survey (pages 275–292): Y. S. Bakhle
Chapter sixteen Modulation of changing Enzyme task via Hypoxia and Its Physiological results (pages 293–311): S. Alex Stalcup, Joel S. Lipset and Robert B. Mellins
Chapter 17 Non?Respiratory features of the Lung within the Perinatal interval (pages 313–331): Robert B. Mellins, Dennis Davidson and S. Alex Stalcup
Chapter 18 Prostaglandin Receptors within the airlines (pages 333–350): H. O. J. Collier and P. J. Gardiner
Chapter 19 The Lung, Whole?Body Metabolism and ailment (pages 351–386): W. A. Stubbs and okay. G. M. M. Alberti
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Additional info for Ciba Foundation Symposium 78 - Metabolic Activities of the Lung
In addition, as seen in both thin-sectioned and freeze-fractured specimens, intercellular junctions of the vascular endothelium vary characteristically from one segment to another in the microvasculature. The intercellular sealing is strong and elaborate in arterioles and is formed by tight junctions with intercalated communicating (gap) junctions. In capillaries the endothelium is provided with continuous, tight junctions only. In postcapillary (pericytic) venules there are only tight junctions but they differ from capillary junctions in being discontinuous and loosely organized; - 30% of the venular junctions appear in sectioned specimens to be open to a space of 3-6 nm (N.
REID& B. MEYRICK Fishman 1976, Ryan & Ryan 1977, Hales et a1 1978, Stalcup et a1 1979) has not been established for the subacute and chronic adaptation or for the other models of hypertension. The hypoxic response demonstrates the structural lability of the circulation. Structural remodelling is apparent within hours to days. The importance of the acute response to hypoxia to the development of the chronic changes we have described is not yet entirely clear. g. McMurtry et a1 1976).
Simionescu: 1 have found them occasionally in the lung, but I don’t have a morphometric estimate. ALVEOLAR-CAPILLARY UNIT 35 Vane: Vesicles are one way of increasing the total surface area of the endothelium. Another way would be to have protrusions from the cells. Scanning electron micrographs show filamentous protrusions from endothelial cells (see Fig. 4b, p 281). Do you see these? Sirnionescu: We sometimes see protrusions of the endothelium in thinsectioned specimens, but fewer than observed in scanning microscopy.