By C. Richard Cothern (auth.), George W. Ware (eds.)
Global cognizance in medical, business, and governmental commumtIes to strains of poisonous chemical substances in foodstuffs and in either abiotic and biotic environ ments has justified the current triumvirate of specialised courses during this box: finished experiences, swiftly released development studies, and archival documentations. those 3 guides are built-in and scheduled to seasoned vide in foreign conversation the coherency crucial for non duplicative and present development in a box as dynamic and complicated as environmental con tamination and toxicology. formerly there was no magazine or different publica tion sequence reserved completely for the diverse literature on "toxic" chemical substances in our meals, our feeds, our geographical atmosphere, our household animals, our natural world, and ourselves. world wide gigantic efforts and plenty of abilities were mobilized to technical and different reviews of natures, locales, magnitudes, fates, and toxicology of the persisting residues of those chemical substances loosed upon the realm. one of the sequelae of this wide new emphasis has been an inescapable desire for an articulated set of authoritative courses the place you'll be able to anticipate finding the newest very important global literature produced by means of this rising quarter of technological know-how including documentation of pertinent ancil lary legislation.
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Extra info for Reviews of Environmental Contamination and Toxicology: Continuation of Residue Reviews
LEER is the Equilibrium Equivalent Concentration of Radon. This is, for a non-equilibrium mixture of short-lived radon progeny in air, that activity concentration of radon in radioactive equilibrium with its short-lived progeny which has the same potential alpha-energy concentration as the actual non-equilibrium mixture). R. Cothern Other preliminary studies of exposure to radon in residences include Port Hope, Ontario (Lees et al. 1987), and Sweden (Damber and Larsson 1987; Stranden, 1986). These are preliminary studies with no specific conclusions.
80%, and that for nonsmokers as: 40,24, 12, and 20% (Saccomanno et al. 1988). It has been suggested that the site for the lung cancers due to radon progeny is the bifurcations or the carina where one passageway splits into two. This is logically where one would expect dust particles carried in the air in the passageways to lodge. However, it is not clear where most tumors start, because the cancerous tissue is large enough by the time it is found to make its specific origin unclear. Most of the data are from death certificates, and therefore no information exists about tumor location.
Y/ , ,-- '-.. ,' I ,~ MALES 0 I 1 1'\' I t" 1 55 ~ ::J , I ,,' I I I - " I ,-I "~ J IL, I V''\. t\ I ~I~' ~ I l,-'::-. :;! « W ,," ,, 1 I II II 10 20 30 40 50 60 70 80 - - NeRP78 --- - BEIR IV - - IORP50 Fig. 5. Plots of the excess lung cancer incidence per lOO,OOO/year as a function of age at observation for risk for the ICRP50, BEIR IV and NCRP 78 models (from NCRP 1988 with permission). :l1 oa.. 8 1---,----------; a: w a.. d o 8 o a.. j;. , >::j' ...... R. Cothern Table 5. 9 pCi/L). A 50% equilibrium factor was used.